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Combined intramuscular and intraspinal transplant of bone marrow cells  improves neuromuscular function in the SOD1G93A mice | Stem Cell Research &  Therapy | Full Text
Combined intramuscular and intraspinal transplant of bone marrow cells improves neuromuscular function in the SOD1G93A mice | Stem Cell Research & Therapy | Full Text

ALS-Linked SOD1 Mutants Enhance Neurite Outgrowth and Branching in Adult  Motor Neurons
ALS-Linked SOD1 Mutants Enhance Neurite Outgrowth and Branching in Adult Motor Neurons

Optimised and Rapid Pre-clinical Screening in the SOD1G93A Transgenic Mouse  Model of Amyotrophic Lateral Sclerosis (ALS) | PLOS ONE
Optimised and Rapid Pre-clinical Screening in the SOD1G93A Transgenic Mouse Model of Amyotrophic Lateral Sclerosis (ALS) | PLOS ONE

General behavior of SOD1 G93A mice. (A) Body weight variations in... |  Download Scientific Diagram
General behavior of SOD1 G93A mice. (A) Body weight variations in... | Download Scientific Diagram

A human-derived antibody targets misfolded SOD1 and ameliorates motor  symptoms in mouse models of amyotrophic lateral sclerosis | Science  Translational Medicine
A human-derived antibody targets misfolded SOD1 and ameliorates motor symptoms in mouse models of amyotrophic lateral sclerosis | Science Translational Medicine

SOD1-G93A transgenic Mouse Models - QPS Neuropharmacology
SOD1-G93A transgenic Mouse Models - QPS Neuropharmacology

Downregulating carnitine palmitoyl transferase 1 affects disease  progression in the SOD1 G93A mouse model of ALS | Communications Biology
Downregulating carnitine palmitoyl transferase 1 affects disease progression in the SOD1 G93A mouse model of ALS | Communications Biology

Frontiers | Human Motor Neurons With SOD1-G93A Mutation Generated From  CRISPR/Cas9 Gene-Edited iPSCs Develop Pathological Features of Amyotrophic  Lateral Sclerosis
Frontiers | Human Motor Neurons With SOD1-G93A Mutation Generated From CRISPR/Cas9 Gene-Edited iPSCs Develop Pathological Features of Amyotrophic Lateral Sclerosis

Tempol improves neuroinflammation and delays motor dysfunction in a mouse  model (SOD1G93A) of ALS | Journal of Neuroinflammation | Full Text
Tempol improves neuroinflammation and delays motor dysfunction in a mouse model (SOD1G93A) of ALS | Journal of Neuroinflammation | Full Text

Targeting Extracellular Cyclophilin A Reduces Neuroinflammation and Extends  Survival in a Mouse Model of Amyotrophic Lateral Sclerosis | Journal of  Neuroscience
Targeting Extracellular Cyclophilin A Reduces Neuroinflammation and Extends Survival in a Mouse Model of Amyotrophic Lateral Sclerosis | Journal of Neuroscience

Simvastatin accelerated motoneurons death in SOD1G93A mice through  inhibiting Rab7-mediated maturation of late autophagic vacuoles | Cell  Death & Disease
Simvastatin accelerated motoneurons death in SOD1G93A mice through inhibiting Rab7-mediated maturation of late autophagic vacuoles | Cell Death & Disease

Overexpression of MT-I in SOD1 G93A mice normalizes copper... | Download  Scientific Diagram
Overexpression of MT-I in SOD1 G93A mice normalizes copper... | Download Scientific Diagram

Multimodal Actions of Neural Stem Cells in a Mouse Model of ALS: A  Meta-Analysis | Science Translational Medicine
Multimodal Actions of Neural Stem Cells in a Mouse Model of ALS: A Meta-Analysis | Science Translational Medicine

Overexpression of MT-I in SOD1 G93A mice ameliorates ALS-like... | Download  Scientific Diagram
Overexpression of MT-I in SOD1 G93A mice ameliorates ALS-like... | Download Scientific Diagram

Parkin is a disease modifier in the mutant SOD1 mouse model of ALS | EMBO  Molecular Medicine
Parkin is a disease modifier in the mutant SOD1 mouse model of ALS | EMBO Molecular Medicine

Increasing MuSK Activity Delays Denervation and Improves Motor Function in  ALS Mice - ScienceDirect
Increasing MuSK Activity Delays Denervation and Improves Motor Function in ALS Mice - ScienceDirect

Amyotrophic Lateral Sclerosis disease - QPS Austria
Amyotrophic Lateral Sclerosis disease - QPS Austria

Preliminary Observation about Alteration of Proteins and Their Potential  Functions in Spinal Cord of SOD1 G93A Transgenic Mice
Preliminary Observation about Alteration of Proteins and Their Potential Functions in Spinal Cord of SOD1 G93A Transgenic Mice

004435 - SOD1-G93A Strain Details
004435 - SOD1-G93A Strain Details

Neuron-Specific Expression of Mutant Superoxide Dismutase Is Sufficient to  Induce Amyotrophic Lateral Sclerosis in Transgenic Mice | Journal of  Neuroscience
Neuron-Specific Expression of Mutant Superoxide Dismutase Is Sufficient to Induce Amyotrophic Lateral Sclerosis in Transgenic Mice | Journal of Neuroscience

002726 - SOD1-G93A Strain Details
002726 - SOD1-G93A Strain Details

Motor neuronal protection by l-arginine prolongs survival of mutant SOD1 ( G93A) ALS mice - ScienceDirect
Motor neuronal protection by l-arginine prolongs survival of mutant SOD1 ( G93A) ALS mice - ScienceDirect

Distinct roles for motor neuron autophagy early and late in the SOD1G93A  mouse model of ALS | PNAS
Distinct roles for motor neuron autophagy early and late in the SOD1G93A mouse model of ALS | PNAS

SOD1-G93A transgenic Mouse Models - QPS Neuropharmacology
SOD1-G93A transgenic Mouse Models - QPS Neuropharmacology

Molecular Chaperone Mediated Late-Stage Neuroprotection in the SOD1G93A  Mouse Model of Amyotrophic Lateral Sclerosis | PLOS ONE
Molecular Chaperone Mediated Late-Stage Neuroprotection in the SOD1G93A Mouse Model of Amyotrophic Lateral Sclerosis | PLOS ONE

Molecules | Free Full-Text | A Novel Anti-Inflammatory d-Peptide Inhibits  Disease Phenotype Progression in an ALS Mouse Model | HTML
Molecules | Free Full-Text | A Novel Anti-Inflammatory d-Peptide Inhibits Disease Phenotype Progression in an ALS Mouse Model | HTML

Delayed Disease Onset and Extended Survival in the SOD1G93A Rat Model of  Amyotrophic Lateral Sclerosis after Suppression of Mutant SOD1 in the Motor  Cortex | Journal of Neuroscience
Delayed Disease Onset and Extended Survival in the SOD1G93A Rat Model of Amyotrophic Lateral Sclerosis after Suppression of Mutant SOD1 in the Motor Cortex | Journal of Neuroscience